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It is also possible that you have nerve or pulpal damage in the traumatized tooth.
The effect of desalivation upon pulpal function and dental caries in rats.
Attempts are made not to extract a pulpal exposure as to maintain space in the primary dentition.
According to neuronal studies, 70-80% of pulpal axons are unmyelinated.
False-positive responses in teeth may be attributed to pulpal necrosis.
It is caused by pulpal necrosis secondary to dental caries or trauma.
It is a productive pulpal inflammation where the development of granulation tissue is seen.
Although periapical changes will be in response to pulpal changes the majority of the time, it is still important to determine the disease process sequence.
It may mean hardening in the inner core of the tooth (pulpal calcification) has occurred.
This is virtually diagnostic of pulpal infection.
Common problems with some of the current dental materials include chemical leakage from the material, pulpal irritation and less commonly allergy.
Similarly, a periodontal abscess may very well appear to be pulpal in origin, when in fact it is not.
The ultimate effect on pulpal inflammation and healing is dependent upon the integrated actions of these inflammatory mediators.
Formative: cells of the pulp organ produce dentin which surrounds and protects the pulpal tissue.
Therefore, lining is normally not necessary under conventional glass ionomer restorations when there is no pulpal exposure.
It is characterized by presence of normal enamel but atypical dentin with abnormal pulpal morphology.
Throughout life, the amount of pulpal tissue in a tooth decreases, which means that the blood supply to the tooth decreases with age.
Immature DCs are therefore considered to be part of the innate phase of pulpal immune response.
If untreated, CTS can lead to severe pain, possible pulpal death, abscess, and even the loss of the tooth.
The involved tooth is usually vital and has no indication for root canal treatment unless the signs of non-vital or necrotic pulpal tissue were confirmed.
MTA provides a higher incidence and faster rate of reparative dentin formation without the pulpal inflammation.
In another study, the pulpal response to glass ionomer cements in caries-free human premolars planned for extraction was examined.
Internal resorption is an unusual condition of a tooth when the dentin and pulpal walls begin to resorb centrally within the root canal.
During this period, oral trauma and clinically significant infections, including dental caries, periodontal disease, and pulpal infection, should be eliminated.
The resulting pulpal necrosis causes release of toxins at the apex of the tooth leading to periapical inflammation.