thyrotropin-releasing hormone

TRH belongs to a family of several thyrotropin-releasing hormones.

Reduction of thyrotropin-releasing hormone concentrations in central nervous system of African lungfish during estivation.

Thyrotropin-releasing factor (thyrotropin-releasing hormone) has a stimulatory effect on prolactin release.

A stimulation test with thyrotropin-releasing hormone (TRH) is not regarded as useful.

The hypothalamus, in the base of the brain, produces thyrotropin-releasing hormone (TRH).

(T3 and T4) and responds by releasing thyrotropin-releasing hormone (TRH).

TSH is produced when the hypothalamus releases a substance called thyrotropin-releasing hormone (TRH).

Subsequently, T3 is transported into the thyrotropin-releasing hormone (TRH) producing neurons in the paraventricular nucleus.

Thyrotropin-releasing hormone (TRH) stimulates its release; negative feedback of thyroid hormone inhibits it.

The hypothalamus secretes the thyrotropin-releasing hormone that directs the synthesis and secretion of TSH from the pituitary gland.

The first of these factors to be identified are thyrotropin-releasing hormone (TRH) and gonadotropin-releasing hormone (GnRH).

Thyroid-stimulating hormone (TSH), released under influence of hypothalamic Thyrotropin-releasing hormone (TRH)

T3 could then bind to the thyroid hormone receptor in these neurons, and affect the production of thyrotropin-releasing hormone, and thereby regulating thyroid hormone production.

The thyrotropin-releasing hormone receptor (TRHR) is a G protein-coupled receptor which binds the tripeptide thyrotropin releasing hormone.

The TSH production itself is modulated by thyrotropin-releasing hormone (TRH), which is produced by the hypothalamus and secreted at an increased rate in situations such as cold exposure (to stimulate thermogenesis).

Thyrotropin-releasing hormone (TRH) produced by the hypothalamus signals to the pituitary to release thyroid-stimulating hormone (TSH), which then stimulates the thyroid to secrete T and T thyroid hormones.

Hormonal output from the thyroid is regulated by thyroid-stimulating hormone (TSH) produced by the anterior pituitary, which itself is regulated by thyrotropin-releasing hormone (TRH) produced by the hypothalamus.

While many prolactin stimulating and enhancing factors are well known (such as thyrotropin-releasing hormone, oxytocin, and estrogen) those have primary functions other than stimulating prolactin release and the search for hypothetical releasing factor or factors continues.

Peptide hormones and protein hormones consist of three (in the case of thyrotropin-releasing hormone) to more than 200 (in the case of follicle-stimulating hormone) amino acid residues and can have a molecular mass as large as 30,000 grams per mole.

Taltirelin (marketed under the tradename Ceredist) is a thyrotropin-releasing hormone (TRH) analog, which mimics the physiological actions of TRH, but with a much longer half-life and duration of effects, and little development of tolerance following prolonged dosing.

The hypothalamus secretes a number of releasing hormones, often according to a circadian rhythm, into hypophyseal portal system; most of these are stimulatory (thyrotropin-releasing hormone, corticotropin-releasing hormone, gonadotropin-releasing hormone and growth hormone-releasing hormone), apart from dopamine, which suppresses prolactin production.

Imbalance in production of thyroid hormones arises from dysfunction of the thyroid gland itself, the pituitary gland, which produces thyroid-stimulating hormone (TSH), or the hypothalamus, which regulates the pituitary gland via thyrotropin-releasing hormone (TRH).

Prolactin cell (also known as epsilon acidophil, lactotrope, lactotropic cell, lactotroph, mammatroph, mammotroph) is a cell in the anterior pituitary which produces prolactin in response to hormonal signals including dopamine which is inhibitory and thyrotropin-releasing hormone which is secretagogue.