"Vav1 and vav3 have critical but redundant roles in mediating platelet activation by collagen."

The mechanism by which platelet activation occurs remains unknown.

Five mechanisms keep platelet activation and the coagulation cascade in check.

Most consumptive conditions lead to platelet activation, and some are associated with thrombosis.

It is a receptor for fibrinogen and aids in platelet activation.

It causes platelet activation in the blood forming clots and the build up of pressure.

The vascular damage is aggravated by platelet activation and recruitment of granulocytes.

This latter finding provides further evidence of the in vivo platelet activation and release reaction.

The sample is then tested for the release of serotonin, a marker of platelet activation.

It inhibits platelet activation and is also an effective vasodilator.