It has been reported that plasmodia can be made to form logic gates.

The stages of the plasmodium develop into more plasmodia by simple fragmentation; at some point, they decide to go sexual.

Myxomycete plasmodia have also been used to study the genetics of asexual cell fusion.

The Myxomycete plasmodium also has the ability to subdivide and establish separate plasmodia.

In molecular biology, Duffy binding proteins are found in plasmodia.

As the causative agent of malaria, Plasmodia sp.

Since this time much progress has been made in understanding the biology of plasmodia and new antimalarial drugs have been discovered.

It has an "apogamic" life cycle; only minute plasmodia are produced, which have never been seen to undergo sexual fusion.

Coenocytes or plasmodia (formed by nuclear division not being followed by cytokinesis).

Increasing the potential immunity generated against Plasmodia can be achieved by attempting to target multiple phases in the life cycle.

Mature plasmodia can produce fruit bodies under appropriate circumstances, the exact triggers for this process are unknown.

These amoebae can mate if they encounter the correct mating type and form zygotes which then grow into plasmodia.

Conversely, separate plasmodia that are genetically similar and compatible can fuse together to create a larger plasmodium.

The mold first densely filled the space with plasmodia, then thinned the network to focus on efficiently connected branches.

The mechanism of action is not fully understood but it is thought to block oxidative metabolism in Plasmodia.

At that time it was also known that plasmodia (the causal agents of malaria) were able to change form during their different developmental stages.

The amoebae and the plasmodia engulf microorganisms.

Myxomycete plasmodia are multinucleate masses of protoplasm that move by cytoplasmic streaming.

Chloroquine/proguanil or sulfa drug-pyrimethamine combinations should be used in all other Plasmodia infections.

The Apicomplexan family of parasites, exemplified by Plasmodia, are major disease agents of humans.

This is a diverse group including organisms such as coccidia, gregarines, piroplasms, haemogregarines, and plasmodia.

Knowledge that plasmodia were vulnerable to sulphonamides was useful, particularly when they were shown to be protected, like bacteria, by p -aminobenzoic acid.

Fusing plasmodia whose cell cycles were out of phase with each other led to a synchronous mitosis in the next mitotic cycle.

Fleischer B. Editorial: 100 years ago: Giemsa's solution for staining of plasmodia.

A person can look well, but still have the Plasmodium in the liver.

Then they change into a different form of Plasmodium.

Weeks or months later, the Plasmodium can leave the liver to the blood, and the person will get sick again.

The plasmodium is white, becoming pink to red at maturity.

The Plasmodium gets into people by the bites of mosquitoes.

The disease is caused by species of the genus Plasmodium.

The Plasmodium is seen in the red blood cells.

The person is then infected with Plasmodium as a by-product.

Sometimes the blood smear will not show Plasmodium even if the person has malaria.

This species had previously been considered to be Plasmodium cnemidophori.

The life cycle of all Plasmodium species is complex.

The second trophic phase begins with the development of the plasmodium.

Up to 2010, three fruit bodies, two spores and one plasmodium have been described.

But the Plasmodium in the liver is not killed by chloroquine.

This is to kill the Plasmodium in the liver.

The generation of resistance can be complicated and varies between plasmodium species.

His best known publication is probably the description of Plasmodium knowlesi in 1927.

One possible experimental infection has been reported with Plasmodium eylesi.

Five species of Plasmodium can infect and be transmitted by humans.

There are no morphological features to distinguish them from any other Plasmodium species.

A plasmodium is formed from the division of many amoeba-like cells.

Like all Plasmodium species it has vertebrate and insect hosts.

Additional host species can be found under the link Plasmodium relictum.

It has been suggested that the plasmpesin family is smaller in other human Plasmodium species.

It is also seen in the case of human parasite Plasmodium malariae.