A person can look well, but still have the Plasmodium in the liver.

Then they change into a different form of Plasmodium.

Weeks or months later, the Plasmodium can leave the liver to the blood, and the person will get sick again.

The plasmodium is white, becoming pink to red at maturity.

The Plasmodium gets into people by the bites of mosquitoes.

The disease is caused by species of the genus Plasmodium.

The Plasmodium is seen in the red blood cells.

The person is then infected with Plasmodium as a by-product.

Sometimes the blood smear will not show Plasmodium even if the person has malaria.

This species had previously been considered to be Plasmodium cnemidophori.

The life cycle of all Plasmodium species is complex.

The second trophic phase begins with the development of the plasmodium.

Up to 2010, three fruit bodies, two spores and one plasmodium have been described.

But the Plasmodium in the liver is not killed by chloroquine.

This is to kill the Plasmodium in the liver.

The generation of resistance can be complicated and varies between plasmodium species.

His best known publication is probably the description of Plasmodium knowlesi in 1927.

One possible experimental infection has been reported with Plasmodium eylesi.

Five species of Plasmodium can infect and be transmitted by humans.

There are no morphological features to distinguish them from any other Plasmodium species.

A plasmodium is formed from the division of many amoeba-like cells.

Like all Plasmodium species it has vertebrate and insect hosts.

Additional host species can be found under the link Plasmodium relictum.

It has been suggested that the plasmpesin family is smaller in other human Plasmodium species.

It is also seen in the case of human parasite Plasmodium malariae.