An indirect physical effect - such as a change in membrane volume - would therefore be needed to affect the ligand-gated ion channels of nerve cells.

Several ligand-gated ion channels have been linked to some types of frontal and generalized epilepsies.

They are ligand-gated ion channels with binding sites for acetylcholine as well as other agonists.

Conversely, neurotransmission that involves exclusively ligand-gated ion channels is much faster.

It has been suggested that it may be a ligand-gated ion channel.

Ionotropic receptors (also known as ligand-gated ion channels) play an important role in inhibitory postsynaptic potentials.

The prototypic ligand-gated ion channel is the nicotinic acetylcholine receptor.

Capsaicin activates C fibers by opening a ligand-gated ion channel and causing an action potential to occur.

The role of tyrosine phosphorylation in the regulation of ligand-gated ion channels in the central nervous system has been less clear.

NETs have conductances similar to those of ligand-gated ion channels.