insulin release

Diabetes is an inability of the body to regulate insulin release.

The increase in calcium will initiate more insulin release from each beta cell.

Insulin release was significantly the highest after intake of the 14% protein diet.

Whey triggers about the double the amount of insulin release.

They act by increasing insulin release from the beta cells in the pancreas.

Sweet tastes also promote insulin release, which blocks your body's ability to burn fat.

As a consequence, insulin release from the pancreatic beta cells is increased.

Insulin stimulation index shows up to 21-fold increase of insulin release.

Treatment with indomethacin (2 mg/kg) had no significant influence on insulin release.

The resulting calcium influx encourages insulin release from beta cells.

Under the same conditions MH caused a complete block of insulin release.

The effects of purinergic agonists on insulin release are controversial in the literature.

Evidence has accumulated that gut hormones stimulate insulin release from the pancreas.

Diazoxide interferes with insulin release through its action on potassium channels.

Calcium is also thought to participate in insulin release, but the researchers found no link between the mineral and risk of developing diabetes later.

They were also resistant to pargyline, a chemical that normally precipitates insulin release.

As for the first phase, insulin release is triggered rapidly when the blood glucose level is increased.

Caffeine may cause a rise in blood sugar and this, by stimulating insulin release, may lead to fat deposition.

It does not stimulate insulin release and hence does not cause hypos.

Novel pathways in the metabolic regulation of insulin release from pancreatic beta-cells.

This allows for the formation of a subcutaneous depot that results in slow insulin release into the systemic circulation.

This inhibition could be partially overcome by 16 mM glucose but the insulin release was still significantly reduced.

Adrenalin (epinephrine) - inhibition of insulin release by the pancreas. 51.

The insulin release stimulated by glucose in pancreatic islets was not modified after stress.

The mice become overweight, and develop high blood sugar levels and reduced insulin release, as expected before the onset of type 2 diabetes.