Due to temporal summation, a slow-wave potential will periodically reach threshold and generate an action potential.

The threshold value controls whether or not the incoming stimuli are sufficient enough to generate an action potential.

The release of neurotransmitter at a ribbon synapse, in turn, generates an action potential in the connected auditory-nerve fiber.

This depolarization opens some voltage-gated sodium channels, but not enough to generate an action potential.

Heart cells (cardiac myocytes) contract due to an increase in the charge across the membrane (depolarization), which generates an action potential.

This current is generally large enough to generate an action potential, called a delayed after-depolarization.

The bathmotropic effect modifies the heart muscle membrane excitability, and thus the ease of generating an action potential.

If the neuron only gets excitatory impulses, it will also generate an action potential.

This allows the potential to reach the threshold to generate an action potential.

This will increase the neuronal excitability and lower the threshold for generating an action potential.