Lymphocytic infiltration can occur in all exocrine glands, including those of the gastrointestinal mucosa.

Immunohistological investigation shows the same changes in the gastrointestinal mucosa as seen in the salivary glands.

When gastric hydrochloric acid reaches the nerves in the gastrointestinal mucosa, they signal pain to the central nervous system.

It is claimed to have protective effects in the gastrointestinal mucosa and in the liver.

They are stable secretory proteins expressed in gastrointestinal mucosa.

Its mode of action is thought to be its ability to potentiate the defensive barriers of gastrointestinal mucosa.

Fosinopril is de-esterified by the liver or gastrointestinal mucosa and is converted to its active form, fosinoprilat.

Thus, the dietary intake of precursor fatty acids can directly influence the rate and pattern of prostaglandin generation by the gastrointestinal mucosa.

Tartar emetic causes intense nausea, prostration and vomiting by irritating the gastrointestinal mucosa.

C.tertium does not appear to secrete any toxin; instead, it damages gastrointestinal mucosa by direct colonization.