The reason low-dose fetal exposure produces such variable outcome is partially rooted in significant genetic differences between individuals.

There is evidence that this reflects fetal exposure to the hormones testosterone and estrogen.

The uterine cavity has a "T-shape" as a result of fetal exposure to diethylstilbestrol.

Yet other studies point to fetal exposure to environmental toxins like lead, cigarettes and alcohol.

After 30,000 uses to date in the U.S., there has not been one reported case of fetal exposure.

Below is chart that states only a few of the developmental tasks hindered in fetal exposure to enviornmentak toxins.

Toxins include fetal exposure to lead, mercury, and ethanol or hazardous environments.

The new research has found a wide spectrum of ill effects that can result from fetal exposure to cocaine.

Among the more worrisome effects of fetal exposure to cocaine are often-subtle impairments in the development of the nervous system.

Two studies linked neurotoxic effects like delays in mental development to chronic fetal exposure to high levels of mercury from fish.