Benzodiazepines, including flunitrazepam have been shown to act on benzodiazepine binding sites as Ca2+ channel blockers and significantly inhibit depolarization-sensitive calcium uptake in rat brain cell components.

A receptors are widely distributed, and act to inhibit calcium uptake.

This effect is mediated by norepinephrine increasing the calcium uptake and therefore greater force production in the muscle.

MCU is an essential component of the mitochondrial membrane and sliencing MCU abolishes calcium uptake.

The neuroprotective actions may, at least in part, occur by blocking calcium uptake into neurons.

This should not be confused, however, with catecholamine-induced calcium uptake into the sarcoplasmic reticulum, which increases lusitropy.

Do not confuse this calcium mechanism with calcium uptake from the extracellular fluid.

Benzodiazepines acted via micromolar benzodiazepine binding sites as Ca channel blockers and significantly inhibit depolarization-sensitive calcium uptake in experimentation on rat brain cell components.

My concern is that we may be unwittingly setting these students up for a Vitamin D deficiency, which has serious implications in bone density and calcium uptake.

Parathyroid hormone is increased which leads to increases of calcium uptake in the gut and reabsorption by the kidney.