Levels of sucrose and pH also have been shown to have minor effects on bacterial survival.

The addition of 0.5% salt (NaCl) also aided bacterial survival, while the sucrose and pH levels had minor effects, as well.

The Helicobacter pylori pyrB gene encoding aspartate caramoyltransferase is essential for bacterial survival.

If blocking a gene kills the bug, then that gene is deemed essential for bacterial survival and could be a target for a drug.

Homeostasis and regulated uptake for metabolic pathways is essential for bacterial survival.

A large number of P. syringae genes contribute to bacterial survival on and within the host plant, including those involved in bacterial attachment and nutrient uptake.

Virulence-related outer membrane proteins are expressed in Gram-negative bacteria and are essential to bacterial survival within macrophages and for eukaryotic cell invasion.

It has been suggested that lysogeny may generally have a role in bacterial survival in animal hosts, and perhaps in pathogenesis.

There are arguments for creating or maintaining conditions that are not conducive to bacterial survival and multiplication, rather than attempting to kill them with chemicals.

Under this mechanism, dauthter cells are protected from receiving damaged copies of the bacterial chromosome, and at the same time promoting bacterial survival.