This mouse model of Huntington's made it possible to look closely at the abnormal protein during the disease process.

These abnormal extended proteins have been shown to be more rapidly degraded within cells and, thus, produce little current.

The killer T-cells recognize these cells because they have abnormal proteins on their surfaces.

Attention seems to be centering on abnormal proteins, but disagreement remains.

Amyloidosis occurs when abnormal proteins build up and form deposits.

Cardiac amyloidosis, a buildup of an abnormal protein in the heart muscle.

The slices are treated with antibodies that bind to abnormal proteins.

Such abnormal proteins are produced due to mutation of the concerned gene.

Alternatively, the abnormal protein might have other types of molecules, like metals, stuck to it.

But the abnormal protein could be folded in such a way that it is resistant to enzyme breakdown.