tracheobronchitis

Until recently, infectious tracheobronchitis was considered to not be a human health risk.

Several puppies were diagnosed with kennel cough but developed severe tracheobronchitis and pneumonia.

Upper respiratory findings include nasopharyngitis and tracheobronchitis.

In dogs, B. bronchiseptica causes acute tracheobronchitis, which typically has a harsh, honking cough.

Kennel cough or tracheobronchitis is a very easily caught dog illness characterized by inflammation (redness and pain) of the upper airways.

The illness is a type of bronchitis called infectious tracheobronchitis that occurs most frequently in dogs and is characterized by a hacking cough.

Ulcerative and plaque-like tracheobronchitis due to infection with Aspergillus in patients with AIDS.

Invasive aspergillosis in the HIV-infected patient is evidenced most commonly as a respiratory illness that can be a necrotizing pneumonia or a tracheobronchitis (650).

Acute respiratory distress and wheezing or fungal cast production can occur with necrotizing tracheobronchitis, and stridor can occur with laryngotracheitis (214,217,225).

In normal, healthy adults there does not appear to be a risk, but young children and immunocompromised individuals should take precautions against coming into contact with animals that have symptoms of tracheobronchitis.

Other less serious complications of R.S.V. infection in young children include tracheobronchitis, middle ear infection and reactive airway disease, a tendency to wheeze when exposed to any respiratory irritant.

Pseudoephedrine is also indicated for vasomotor rhinitis, and as an adjunct to other agents in the optimum treatment of allergic rhinitis, croup, sinusitis, otitis media, and tracheobronchitis.

Cats infected with B. bronchiseptica have been seen with tracheobronchitis, conjunctivitis and rhinitis (upper-respiratory tract infection or URI), mandibular lymphadenopathy, and pneumonia.

Other manifestations include necrotizing tracheobronchitis; pseudomembranous tracheobronchitis; and involvement of CNS, skin, sinuses, middle ear, and mastoid bones (217--221,227).

Moraxella catarrhalis usually resides in respiratory tract, but can gain access to the lower respiratory tract in patients with chronic chest disease or compromised host defenses, thus causing tracheobronchitis and pneumonia.

Signs of pulmonary toxicity begins with evidence of tracheobronchitis, or inflammation of the upper airways, after an asymptomatic period between 4 and 22 hours at greater than 95% oxygen, with some studies suggesting symptoms usually begin after approximately 14 hours at this level of oxygen.