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This relatively slow depolarization continues until the threshold potential is reached.
The electrical potential at which this occurs is called the threshold potential.
This specific value of depolarization (in mV) is otherwise known as the threshold potential.
As opposed to the resting membrane potential, the threshold potential's conditions exhibited a balance of currents that were unstable.
Most often the threshold potential is a membrane potential value between -40 and -55 mV, but it can vary based upon several factors.
If the stimulus exceeds the threshold potential, the nerve or muscle fiber will give a complete response; otherwise, there is no response.
As a result, the difference between resting potential and threshold potential is increased and firing is less likely.
That is, the maximum diastolic potential is less negative and therefore exists closer to the threshold potential.
All afterdepolarisations may not reach threshold potential, but, if they do, they can trigger another afterdepolarisation, and thus self-perpetuate.
The threshold potential is the critical level to which the membrane potential must be depolarized in order to initiate an action potential.
The firing of the pacemaker cells is induced electrically by reaching the threshold potential of the cell membrane.
Threshold potential ("All or nothing")
As ischemia occurs through inhibition of the sodium-potassium pump, abnormalities in the threshold potential are hence implicated.
At each successive node, the membrane potential of the axon is thereby brought to the threshold potential to initiate an action potential.
The threshold potential is the potential an excitable cell membrane, such as a myocyte, must reach in order to induce an action potential.
By inhibiting the voltage-dependent sodium current, these oils shift the threshold potential to a more positive value; therefore, an action potential will require increased depolarization.
There is some indirect evidence that the caudate may perform this regulatory role by measuring the general activity of cerebral cortex and controlling the threshold potential.
With stochastic resonance, synaptic noise can amplify the recognition of signals that are below threshold potential in nonlinear, threshold-detecting systems.
Since the experiment yielded results through the observation of ionic conductance changes, Hodgkin and Huxley used these terms to discuss the threshold potential.
They soon discovered that at threshold potential, the inward and outward currents, of sodium and potassium ions respectively, were exactly equal and opposite.
Furthermore, it can be used to identify characteristics of significant medical conditions through comparing the effects of those conditions on threshold potential with the effects viewed experimentally.
This increase in membrane potential typically permits the membrane potential to reach the threshold potential at which it fires the next action potential (Pacemaker potential).
As various drugs and other factors act on the resting potential and bring it closer to the threshold potential, the action potential is more easily and rapidly obtained.
Depolarization of the membrane past its Threshold potential generates an action potential, which is the main source of signal transmission, known as Neurotransmission of the nervous system.
At the axon hillock of a typical neuron, the resting potential is around -70 millivolts (mV) and the threshold potential is around -55 mV.