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These receptors are used to pass on an action potential across the synaptic cleft between neurons.
This 30-nanometer space forms the synaptic cleft through which signalling molecules are released.
This increases the availability of 5-HT in the synaptic cleft.
This interaction seems to be critical for increased availability of norepinephrine in or near the synaptic clefts.
When the acetylcholine is not degraded, it accumulates in the synaptic cleft.
Acetylcholine is released from presynaptic neurons into the synaptic cleft.
Some drugs inhibit the reuptake of serotonin, making it stay in the synaptic cleft longer.
This occurs only when the concentration of ACh in the synaptic cleft is very low.
These molecules then bind to receptors on the postsynaptic cell's side of the synaptic cleft.
Some models are also tracking biochemical pathways at very small scales such as spines or synaptic clefts.
Between the pre- and postsynaptic cells is a gap about 20 nm wide called the synaptic cleft.
The neurotransmitter molecules diffuse across the synaptic cleft to the postsynaptic membrane.
The presence of the pore allows for the release of neurotransmitter into the synaptic cleft.
"Well, right now I'm working on the dopamine and noradrenaline reactions-attempting to extend the time the stuff remains in the synaptic cleft."
Excess monoamine neurotransmitters in the synaptic cleft leads to overstiumulation of the cells.
Furthermore, they do not cross the synaptic cleft, and therefore only label the neurons that were directly at the site of tracer application.
Acetylcholine diffuses across the synaptic cleft and binds to the muscle fiber membrane.
Acetylcholine then diffuses across the synaptic cleft.
Then, the neurotransmitter is reabsorbed from the synaptic cleft into the cytoplasm of the neuron.
A possible explanation for this long lasting behavior might be that muscarine does not get hydrolyzed by acetylcholinesterase in the synaptic cleft.
The mode of action is a strong reuptake inhibition of serotonin from the synaptic cleft.
The reuptake of these two neurotransmitters is essential in regulating concentrations in the synaptic cleft.
Synaptic Cleft - A real-time game of forces where objects enter from the left and must be guided to a small aperture on the right.
Calcium enters the axon terminal during an action potential, causing release of the neurotransmitter into the synaptic cleft.
Perhaps the slender filaments meshed with her synaptic clefts were getting screwed by her disturbed body chemistry?