suppressor T cell

In laboratory experiments, beta interferon brings the suppressor T cell number back to normal.

According to one theory, the suppressor T cells reappear within a month joined by another, more primitive type of suppressor cell.

First, the drug corrects an abnormality in suppressor T cells, which are white blood cells that dampen the immune system.

The cyclophosphamide and epirubicin were administered to block the activity of suppressor T cells that might weaken the desired immune responses.

It is also possible that perturbation of suppressor T cell populations by Campath-1H may precipitate autoimmune disease.

Suppressor T cells are also known as CD25-Foxp3 regulatory T cells (nTregs), and reduce inflammation.

Regulatory T cells (T cells), formerly known as suppressor T cells, are crucial for the maintenance of immunological tolerance.

Additional suppressor T cell populations include Tr1, Th3, CD8CD28, and Qa-1 restricted T cells.

Studies of human subjects with a history of leishmania infection suggest that modulation of CD8+ suppressor T cells is, at least partly, mediated by cytokines.

Soluble factors generated by antigen stimulation of peripheral blood mononuclear cells from skin-test positive adults prevented CD8+ suppressor T cell mediated increases in interleukin-10 secretion.

In immunocompetent patients, Epstein-Barr virus causes infectious mononucleosis, characterised by a proliferation of B-lymphocytes which is controlled by Suppressor T cells.

As a result, the combination prevents alloimmunization, up-regulates Regulatory T cells (suppressor T cells) and reduces the severity of GVHD.

There are three different forms of T cells, which are the Helper T cells, the Killer T cells, and the Suppressor T cells.

These are killer T cells, which would attack the new organ, helper T cells, which would coordinate that attack, and suppressor T cells, which would end it.

One of the prominent autoimmune regulatory mechanisms is a sub-population of T cells called 'regulatory T cells' (previously known as 'suppressor T cells'), which restrict autoimmune activity.

This paradox resulted in suppressor T cells and tabs, which both express I-J determinants, falling out of favour, together with the symmetrical network theory, that is based on the existence of tabs.

An intact sympathetic nervous system is required to maintain full cellular immunoregulation as denervated mice do not produce and activate, for example, splenic suppressor T cells, or thymic NKT cells.

The helper T cells and suppressor T cells that are co-selected are then a mutually stabilizing construct, and for a given mouse genome, more than one such mutually stabilizing set can exist.

While having no direct anti-tumour effect on the cells in vitro, it was found that cimetidine inhibited suppressor T cells allowing the initial anti-tumour response affecting cytotoxic T cells to persist and be effective.

Regulatory T cells (T), formerly known as suppressor T cells (T), are a subpopulation of T cells which modulate the immune system, maintain tolerance to self-antigens, and abrogate autoimmune disease.

One of the mechanisms postulated to play a part in the anti-tumour effect of cimetidine is immunomodulation mediated by the inhibition of suppressor T cell activity, an increase in interleukin 2 production inhelper T cells, and an enhancement of natural killer cell activity.

They found consistent stress-related increases in numbers of total white blood cells, as well as decreases in the numbers of helper T cells, suppressor T cells, and cytotoxic T cells, B cells, and Natural killer cells (NK).