subinhibitory

Biofilms formed in the presence of subinhibitory concentrations of dicloxacillin contain less biomass and have an altered composition.

Bacterial surface hydrophobicity can be altered significantly through exposure to subinhibitory concentrations of antibiotics, surfactants, lectins, etc.

The fission yeast Schizosaccharomyces pombe utilizes acetate at subinhibitory concentrations in the presence of D-glucose.

Subinhibitory concentrations (sub-MIC) of the antibiotic dicloxacillin also affect the growth of S. haemolyticus biofilms.

As a result, studies were able to conclude that the subinhibitory nature of vancomycin and penicillin levels were increasing the toxin production in cultures of C. difficile.

The effects of subinhibitory doses of chloramphenicol and oxytetracycline on chromogen formation by normal and antibiotic-resistant Pseudomonas aeruginosa strains were studied.

The effect of subinhibitory concentrations of seven antimicrobials on meningococcal adherence, piliation, hemagglutination (HA), and bacterial proteins was studied to determine their potential for modifying virulence.

Strains of Pseudomonas aeruginosa exposed to subinhibitory concentrations of either polymyxin B, gentamicin, or carbenicillin were compared with untreated organisms for changes in sensitivity to pyocin.

The superoxide generators paraquat and menadione at subinhibitory concentrations for growth were potent inducers (8.3 and 10.4 fold, respectively) of the catalase of X. oryzae pv. oryzae.

Over 100 strains of N. gonorrhoeae, most of them resistant to penicillin, were screened for their ability to deactivate penicillin during 48 h of growth in the presence of subinhibitory levels.

These results indicate that subinhibitory antibiotic concentrations reduce meningococcal piliation and alter other bacterial proteins; these changes are associated with diminished adherence and hemagglutination, alterations which may be markers of meningococcal virulence.

Subinhibitory concentrations of quinolones, NFLX, sparofloxacin (SPFX), and grepafloxacin (GPFX) markedly stimulated the productions of VT1 and VT2.

Capillary electrophoresis revealed only a subinhibitory amount of AMP and no detectable GMP, and addition of AMP or GMP to this medium also reduced competence 20-100-fold.