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Research has shown that mir-143 plays an important role in smooth muscle cell fate.
Eventually smooth muscle cells proliferate at the site of injury.
These include the smooth muscle cells of the arteries.
Several theories of the factors causing smooth muscle cell proliferation have been made.
Smooth muscle cells can be stimulated to contract or relax in many different ways.
It is a benign neoplasm composed of smooth muscle cells.
The smooth muscle cells were plated on to P-100's and grown to confluence.
Leiomyosarcoma starts in the smooth muscle cells of the small intestine.
These effects can be explained by the relaxing activity of calciseptine on various smooth muscle cells.
This protein is involved in the regulation of vascular smooth muscle cell proliferation.
Tunica media is made up of smooth muscle cells and elastic tissue.
The Bayliss effect in vascular smooth muscles cells is a response to stretch.
The smooth muscle cells are able to constrict from both external and internal stimuli.
Action of catecholamines on the smooth muscle cell membrane.
These smooth muscle cells have M3 type muscarinic receptors on their membrane.
Myofibroblasts contain the same kind of actin as that found in smooth muscle cells.
The myofibrils of smooth muscle cells are not arranged into sarcomeres.
It occurs to a lesser extent in bone, cartilage, and smooth muscle cells.
Its target channels can be found in cardiac tissue, neurons and smooth muscle cells.
The media is made up of smooth muscle cells that are flexible, allowing the aorta to expand and contract.
Myofibroblasts, which are similar to smooth muscle cells, are responsible for contraction.
The calcium blockers relax smooth muscle cells in blood vessel walls, lowering the pressure.
Smooth muscle cells start to grow and proliferate, forming a tumor-like bulge.
It is useful in the study of small nerve fibers and electrically connected cells such as smooth muscle cell.
It is suggested that Kv1.3 is important in proliferating vascular smooth muscle cells.