regulator gene

The regulator gene is always 'on', and synthesizes repressor at a very low rate.

A question of interest for biologists studying cell identity is what regulates the master regulator genes.

This inhibition would further progress the malignancy of the cells since they lose key regulator genes.

They found it had sought out some 200 sites where many of the master regulator genes of human cells are located.

There will also be regulator genes.

Regulator genes can be located within an operon, adjacent to it, or far away from it.

The Fox genes are a large family of master regulator genes that control other genes.

In prokaryotes, regulator genes often code for repressor proteins.

The Broad team then looked at the chromatin state of the master regulator genes in several kinds of mature cell.

Myc (c-Myc) is a regulator gene that codes for a transcription factor.

The regulator gene is araC.

The developmental programs, directing a cell to become a neuron, say, or a liver cell, are initiated by master regulator genes.

Repressor proteins are produced by a regulator gene, but they are unable to bind to the operator in their normal conformation.

They have also cloned and characterized the fur regulator gene of the IROMPs.

A regulator gene, regulator, or regulatory gene is a gene involved in controlling the expression of one or more other genes.

The CFTR locus is named for one of the 10 genes it contains, the cystic fibrosis transmembrane conductance regulator gene.

Cystic fibrosis (caused by the G542X mutation in the cystic fibrosis transmembrane conductance regulator gene).

Cystic fibrosis, a congenital disorder which affects about one in 2000 children, is caused by a mutant form of the CF transmembrane regulator gene, CFTR.

A regulator gene may encode a protein, or it may work at the level of RNA, as in the case of genes encoding microRNAs.

Although only half of the highly conserved regions contain master regulator genes, something in their DNA structure may be the signal that tells the cell where to create the bivalent domains.

One study found that about a quarter of synonymous variations affecting exon 12 of the cystic fibrosis transmembrane conductance regulator gene result in that exon being skipped.

Dr. Young showed last year that the genes make transcription factors that act on each other's control sites in ways that in effect form a circuitry for controlling the master regulator genes.

Each cell must avoid being committed to any particular fate for the time being, so all its master regulator genes must be repressed by tight winding of the spools that hold their DNA.

A mutation of the cystic fibrosis transmembrane conductance regulator gene causes defective CFTR proteins, which are transmembrane proteins that function in Cl- transport in wet epithelia.

A regulator gene marR was mutated and a normally non-translated membrane protein yedS was expressed, both were demonstrated to have effects on the ability of this strain of E.coli to resist carbapenems.