postsynaptic membrane

This may make the postsynaptic membrane depolarized (less negative).

These neurotransmitters bind to receptors on the postsynaptic membrane and lead to its depolarization.

These small depolarizations are not enough to reach threshold and so an action potential in the postsynaptic membrane does not occur.

These receptors are involved in presynaptic inhibition, and do not appear to affect postsynaptic membrane potential by themselves.

On the postsynaptic membrane, neural noise has been evident in the early stages of processing sight, smell, and hearing.

This spike causes an action potential to occur and propagate down the postsynaptic membrane leading to muscle contraction.

The neurotransmitter molecules diffuse across the synaptic cleft to the postsynaptic membrane.

Nicotinic acetylcholine receptors are positioned in the postsynaptic membrane of nerve cells.

After its release, the transmitter binds to and activates a receptor in the postsynaptic membrane.

As with PKCβ, gravin was seen to be directed to the postsynaptic membrane.

An electrical current is generated which changes the postsynaptic membrane potential to create a more negative postsynaptic potential.

The neurotransmitter then binds to receptors embedded in the postsynaptic membrane, causing them to enter an activated state.

Synaptosomes isolated from mammalian brain often retain a piece of the attached postsynaptic membrane, facing the active zone.

EPP are caused mostly by the binding of acetylcholine to receptors in the postsynaptic membrane.

For example, curare is a poison that stops acetylcholine from depolarizing the postsynaptic membrane, causing paralysis.

This restriction to the membrane allows it to bind to and cluster ion channels in the postsynaptic membrane.

Inhibition of this enzyme causes a constant stimulation of the postsynaptic membrane by the neurotransmitter which it cannot cancel.

The subsequent change in stimulation threshold of the postsynaptic membrane can be enhanced or inhibited, depending on the transmitter chemical involved and the ion permeabilities.

Acetylcholine binds to acetylcholine receptors at the postsynaptic membrane to open the sodium-channels there, generating a new action potential.

The drug blocks the active site of acetylcholinesterase so the enzyme can no longer break down the acetylcholine molecules before they reach the postsynaptic membrane receptors.

The motor neuron then innervates the muscle fibers to contraction by causing an action potential on the postsynaptic membrane of the neuromuscular junction.

It has also been reported that the short insectotoxin I5A may act on a "glutamate receptor of the postsynaptic membrane" [ 22 ] .

PKCβ colocalizes with gravin at the postsynaptic membrane where the gravin staining is strongest.

The postsynaptic density (PSD) is a protein dense specialization attached to the postsynaptic membrane.

Second, they mediate or modulate the insertion of additional AMPA receptors into the postsynaptic membrane.