plasma osmolality

The two factors work together to return the plasma osmolality to its normal levels.

This measurement gives the true plasma osmolality.

This is called an increase in plasma osmolality, and it is detected by a part of our brain called the hypothalamus.

Therefore, plasma osmolality is a guide to intracellular osmolality.

Plasma osmolarity can be calculated from plasma osmolality by the following equation:

Any significant rise in plasma osmolality is detected by the hypothalamus, which communicates directly with the posterior pituitary gland.

By doing so, the orignal signal for action is eventually removed (you restore your plasma osmolality to normal) and you are no longer thirsty.

Hypoosmolar hyponatremia is a condition where hyponatremia associated with a low plasma osmolality.

Otherwise, one litre of plasma would be equivalent to one kilogram of plasma, and plasma osmolarity and plasma osmolality would be equal.

Antidiuretic hormone (ADH) stimulates the kidney tubules to reabsorb and conserve water, resulting in small volumes of highly concentrated urine and decreased plasma osmolality.

This was revealed as increased levels of AVPR1B mRNA on the choroid plexus were discovered as a result of increased plasma osmolality.

This dilution, otherwise described as a reduction in plasma osmolality, is detected by osmoreceptors in the hypothalamus of the brain and these then switch off the release of ADH.

The maintenance of our bodies fluid volume and the concentration of the this fluid (called the plasma osmolality) is regulated by our water/liquid intake and kidney output of water and sodium.

Sensors detect an imbalance (high plasma osmolality) and cause the release and activation of effectors (thirst sensation, drinking behavior, angiotensin 2 and ADH release) that restore proper balance.

In SIADH the release of ADH is not inhibited by a reduction in plasma osmolality when the individual ingests water and the osmolality of the plasma drops.

Vasopressin is secreted from the posterior pituitary gland in response to reductions in plasma volume, in response to increases in the plasma osmolality, and in response to cholecystokinin (CCK) secreted by the small intestine: