The osteoblast creates and repairs new bone by actually building around its self.

The brand new bone created by the osteoblast is called osteoid.

These alloys were found to cause neoplastic transformations of human osteoblast cells.

A deficient osteoblast layer and widespread external resorption is also observed.

Recent research has suggested that certain growth factors may work to locally alter bone formation by increasing osteoblast activity.

Dose-dependent effects of strontium on osteoblast function and mineralization.

Activated caspase-8 was detected in uninfected control osteoblast cultures (Fig.

First, the osteoblast puts up collagen fibers.

When the osteoblast becomes trapped, it becomes known as a osteocyte.

RUNX2 is a key transcription factor associated with osteoblast differentiation.

The release of these growth factors from the bone matrix could cause the proliferation of osteoblast precursors.

Melatonin promotes osteoblast differentiation and bone formation.

Osteoporosis is also an issue in Cushing's syndrome since osteoblast activity is inhibited.

The osteoblast then deposits calcium phosphate which is hardened by hydroxide and bicarbonate ions.

Approximately 14.89% of the cells from uninfected osteoblast cultures were annexin V positive.

S. aureus induces osteoblast apoptosis [ 34 ] .

At various times following bacterial invasion, osteoblasts were lysed and total osteoblast protein isolated for further analysis.

"Osteoblast and chrondroblast differentiation."

The porous bone-like properties of the metal foam contribute to extensive bone infiltration, allowing osteoblast activity to take place.

In contrast, Osteoblast promotes calcium absorption by the bone therefore, promoting bone mineralization and formation of new bones.

BMP4 and FGF2 have been experimentally shown to increase osteoblast differentiation.

An active osteoblast is characterized morphologically by a prominent Golgi apparatus that appears histologically as a clear zone adjacent to the nucleus.

Once the osteoblast is finished working it is actually traped inside of the bone once it hardens.

By a lesser understood mechanism, thiazides directly stimulate osteoblast differentiation and bone mineral formation, further slowing the course of osteoporosis.

Femarelle was found to regenerate the bone through increased osteoblast activity, which makes it a possible agent for postmenopausal bone loss.