obesogenic

"Obesogenic environment" is the medical term set aside for this mixture of elements.

In today's obesogenic environment food is abundant and rewarding.

This abnormal regulation leads to a decreased level of androgen in the body which, itself is obesogenic.

Political debate arises over whether obesity is an outcome of individual behavior or a consequence of an increasingly obesogenic environment.

There are many different ways in which obesogenic drugs and chemicals can act in order to disrupt the body's adipose tissue biology.

Sticking with moderate food portions can help you avoid eating too many calories in what researchers call our "obesogenic" environment of super-sized meals and sedentary lifestyles.

Many neuropharmacueticals used to alter behavioral pathways in patients with neurological disorders have shown to have metabolic altering side-effects leading to obesogenic phenotypes as well.

However since obesogenic phenotypes persist, it can be concluded that adaptive response components of the hypothalmus may be a target of obesogens as well.

School Nutrition and Physical Activity Policies, Obesogenic Behaviors and Weight Outcomes (R21)

Both obesogenic drugs and chemicals have been shown to target transcription regulators found in gene networks that function to control intracellular lipid homeostasis and proliferation and differentiation on adipocytes.

Price incentives from beverage companies have maintained their products very cheap This obesogenic environment has promoted the consumption of sugar-sweetened beverages on a national level correlating with trends in the obesity epidemic.

Obesogens detection in the body and resulting obesogenic effects can result as side effects from intentional administration of obesogenic chemicals in the form of pharmaceutical drugs.

Obesogens that target the PPARγ/RXR complex mimic the metabolic ligands and activate the receptor leading to upregulation of lipid accumulation which explains their obesogenic effects.

While obesogens can be introduced to the body intentionally via administration of obesogenic pharmaceuticals, exposure can also occur through chemical exposure to obesogens found in the environment such as organotins and xenobiotics.

Organotins (tin-based chemicals), used in marine anti-fouling paints, wood catalysts, plasticizers, slimicides, in industrial water systems, and fungicides on food have recently been linked to obesogenic properties when introduced in the body.

Some believe it has to do with the "population being a statistical artifact, some believe that childhood obesity is due to compositional factors, and then others believe that the problems are due to something called an "obesogenic environment."

It has been shown that obesogenic effects are apparent in female mice exposed to both phytoestrogens and DES during their neonatal periods of development, as they, though born with a lower birth weight, almost always developed obesity, high leptin levels, and altered glucose response pathways.

BPA from polycarbonate plastics, phthalate plasticizers used to soften PVC plastics, and various perfluoroalkyll compounds (PFCs) that are widely used surfactants and surface repellents in consumer products are all potentially obesogenic when introduced in the body.