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Defects in the genes encoding for nuclear lamin (such as lamin A and lamin B) have been implicated in a variety of diseases (laminopathies) such as:
More recently, using Xenopus egg extracts, it was possible to demonstrate the mitosis-specific function of the nuclear lamin B in regulating spindle morphogenesis () and to identify new proteins that mediate kinetochore attachment to microtubules.
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