neuromuscular junction

Nerves excite muscles in a similar way at the neuromuscular junction.

The space between the nerve terminal and the muscle cell is called the neuromuscular junction.

Evidence for this comes from the neuromuscular junction.

LRP4 plays an important role in the formation of neuromuscular junctions.

Neuromuscular junction diseases can be of genetic and autoimmune origin.

In mice that are deficient for either protein, the neuromuscular junction does not form.

Actions on the neuromuscular junction will result in prolonged muscle contraction.

This axon then forms a neuromuscular junction with the effectors.

The neuromuscular junction is where a neuron activates a muscle to contract.

Additional antibodies against neuromuscular junction channels and receptors have also been described.

The area where the nerve contacts the muscle is called the neuromuscular junction, or the motor end plate.

At the neuromuscular junction, the chemical and electrical aspects of the nerve impulse arrive.

Studies focus on neuromuscular junctions and modulation of abdominal motor cells.

By doing so, they increase the action potential threshold in the neuromuscular junction, eliciting paralytic behavior.

WNT4 contributes to the formation of the neuromuscular junction in vertebrates.

Much of our understanding of synapse formation comes from studies at the neuromuscular junction.

Several motorneurons compete for each neuromuscular junction, but only one survives until adulthood.

A quantitative description at the frog neuromuscular junction.

This gene codes for a voltage-gated sodium channel Na1.4 found at the neuromuscular junction.

If there is a problem at the neuromuscular junction, a very prolonged contraction may occur, tetanus.

It occurs in neuromuscular junction disorders, such as myasthenia gravis.

This includes, but is not limited to, disorders of peripheral nerves, muscle and neuromuscular junctions.

Neuromuscular junction of the rat, mouse and frog.

Toxins are also used to determine the location of acetylcholine receptors at the neuromuscular junction.

This technique proved to be an important model for further studies to determine the essential mechanisms controlling neuromuscular junction development and maintenance.