nephrogenic diabetes insipidus

Most commonly, nephrogenic diabetes insipidus develops because of other reasons.

Call your health care provider if you have symptoms of nephrogenic diabetes insipidus.

There is no known way to prevent congenital nephrogenic diabetes insipidus.

Nephrogenic diabetes insipidus: The kidneys lose the ability to concentrate the urine, usually due to a drug reaction.

Nephrogenic diabetes insipidus that is present at birth is a chronic condition requiring lifelong treatment.

Mutations in the aquaporin-2 gene cause hereditary nephrogenic diabetes insipidus in humans.

Nephrogenic diabetes insipidus is due to the inability of the kidney to respond normally to vasopressin.

Nephrogenic diabetes insipidus is rare.

Mutations in this channel are associated with nephrogenic diabetes insipidus, which can be autosomal dominant or recessive.

Demeclocycline is unique in that it is the only tetracycline known to cause nephrogenic diabetes insipidus.

Effectively, it causes iatrogenic nephrogenic diabetes insipidus.

Instead, the diuretic hydrochlorothiazide (a thiazide diuretic) or indomethacin can improve nephrogenic diabetes insipidus.

In contrast, there is no change if the kidneys are unresponsive to ADH due to a different problem ("nephrogenic diabetes insipidus").

Polyuria/polydipsia (nephrogenic diabetes insipidus)

Differential diagnosis includes nephrogenic diabetes insipidus, neurogenic/central diabetes insipidus and psychogenic polydipsia.

Because lithium competes with the receptors for the antidiuretic hormone in the kidney, it increases water output into the urine, a condition called nephrogenic diabetes insipidus.

Dehydration in patients taking lithium salts can be very hazardous, especially when combined with lithium induced nephrogenic diabetes insipidus with polyuria.

Nephrogenic diabetes insipidus occurs when the kidney tubules do not respond to a chemical in the body called antidiuretic hormone (ADH), also called vasopressin.

Functional tubule abnormalities such as nephrogenic diabetes insipidus result from marked reduction in vasa recta blood flow, combined with ischemic tubule injury.

The second common type of DI is nephrogenic diabetes insipidus, which is caused by an insensitivity of the kidneys to ADH.

In the absence of diabetes mellitus, the most common causes are primary polydipsia (excessive fluid drinking), central diabetes insipidus and nephrogenic diabetes insipidus.

Nephrogenic diabetes insipidus is a disorder in which a defect in the small tubes (tubules) in the kidneys causes a person to pass a large amount of urine.

Congenital nephrogenic diabetes insipidus (NDI) may result from VR or aquaporin-2 (AQP2) mutations.

The use in SIADH actually relies on a side effect; demeclocycline induces nephrogenic diabetes insipidus (dehydration due to the inability to concentrate urine).

Clinicians have been aware of lithium toxicity for many years, and traditionally have administered thiazide diuretics for lithium-induced polyuria and nephrogenic diabetes insipidus.