macrocytic

Many affected children develop anemia, which may be macrocytic in type.

They were enlarged, with unusual structures, described as macrocytic.

Some may also be afflicted with macrocytic anemia.

She conducted seminal work in India in the late 1920s and early 1930s on macrocytic anaemia of pregnancy.

After her return to England, there were a number of further papers concerning her work on tropical macrocytic anaemia.

Defined as those seen in any macrocytic, megaloblastic anemia:

Workers exposed to high levels are at risk for granulocytopenia, macrocytic anemia, oligospermia, and azoospermia.

While heterozygotes are healthy, homozygous W mice have severe macrocytic anemia and die within days.

She postulated that there must have been another nutritional factor responsible for this macrocytic anaemia other than vitamin B deficiency.

Anemia is classified as microcytic or macrocytic based on whether this value is above or below the expected normal range.

In pernicious anemia (macrocytic), MCV can range up to 150 femtolitres.

As expected, a defect at any point of the aforementioned list, can cause malabsoption of vitamin B12, and subsequent macrocytic anemia.

In adults, anemia (macrocytic, megaloblastic anemia) can be a sign of advanced folate deficiency.

Lucy Wills decided to investigate possible nutritional treatments by first studying the effects of dietary manipulation on a macrocytic anaemia in albino rats.

In a macrocytic anemia the larger red cells are always associated with insufficient numbers of cells and often also insufficient hemoglobin content per cell.

The anemia is called "microcytic" if red cells are small, "macrocytic" if they are large, and "normocytic" otherwise.

In 1928 Lucy Wills began her seminal research work in India on macrocytic anaemia in pregnancy.

The characteristic hematological (blood) effects of copper deficiency are anemia (which may be microcytic, normocytic or macrocytic) and neutropenia.

Changes in red blood cell parameters of 20,000 and 50,000 ppm males were consistent with mild macrocytic normochromic anemia with a depressed regenerative response.

Vitamin B12 and/or folic acid deficiency has also been associated with macrocytic anemia (high MCV numbers).

Megaloblastic anemia, the most common cause of macrocytic anemia, is due to a deficiency of either vitamin B, folic acid, or both.

Megaloblastic erythroid hyperplasia with macrocytic anemia, which is associated with normal vitamin B12 and folate levels, is frequently observed.

Vitamin B deficiency produces a macrocytic anemia (large cell anemia) with a high RDW.

Diseases associated with folic acid deficiency include: megaloblastic or macrocytic anemia, cardiovascular disease, certain types of cancer, and NTDs in infants.

For example, macrocytic hypochromic, due to hookworm infestation leading to not enough of both iron and vitamin B12 or folic acid or following a blood transfusion.