intrauterine hypoxia

In many cases, intrauterine hypoxia results in the death of the fetus.

Rubella, intrauterine hypoxia and hypothyroidism are some of the more researched examples.

A response to chronic intrauterine hypoxia.

There are various causes for intrauterine hypoxia (IH).

In the United States intrauterine hypoxia and birth asphyxia was listed as the tenth leading cause of neonatal death.

Intrauterine hypoxia (IH, and birth asphyxia) occur when the fetus is deprived of an adequate supply of oxygen.

Intrauterine hypoxia is a condition or state caused by insufficient oxygen levels reaching a fetus during gestation, having detrimental effects on the development of its central nervous system (CNS).

Intrauterine hypoxia and birth asphyxia can cause hypoxic ischemic encephalopathy which is cellular damage that occurs within the central nervous system (the brain and spinal cord) from inadequate oxygen.

Intrauterine hypoxia or birth asphyxia IH/BA was the ninth most expensive medical condition treated in U.S. hospitals by average hospital cost and resultant hospital charge.

The majority of the research done regarding fetal brain development, and consequently its memory after birth, has focused on one condition or state and two main diseases: intrauterine hypoxia, hypothyroidism and rubella.

Most accounts on the implications caused by diseases or conditions like rubella, hypothyroidism and intrauterine hypoxia involving humans have come from studies done on patients later in life, or more appropriately, after birth.

Intrauterine hypoxia - lack of oxygen - can be both a reason for performing a Caesarean section and a cause of death, but even eliminating those deaths left a neonatal mortality rate for Caesarean deliveries in the cases studied at more than twice that for vaginal births.