hypofrontality

There is not yet an effective treatment for hypofrontality.

Hypofrontality is thought to contribute to the negative symptoms of schizophrenia.

The contribution that hypofrontality has in each case is hard to determine, mostly because the disorders themselves are not fully understood.

Currently, the exact neurophysiological mechanisms that cause hypofrontality are unknown.

This suggests that the mechanisms that cause hypofrontality and these neurological conditions are likely to be similar.

Even with the large amounts of research on hypofrontality in schizophrenia, there is still a lot to be learned about its neuronal mechanisms.

However, there are some possible mechanisms that are plausible and would account for many of the effects of hypofrontality.

Hypofrontality is a condition that is symptomatic of many neurological disorders and psychiatric diseases.

This means that hypofrontality is likely to have causes that stem from bodily changes or changes in neurons.

Dr Jung describes the phenomenon as "transient hypofrontality."

Mostly in the frontal cortex, these differences often stem from a smaller brain volume, and the decreased blood flow that results influences the hypofrontality.

Hypofrontality likely has pathophysiological mechanisms and neuronal mechanisms.

Even though there is still a lot to be learned, experiments on NK1R -/- mice has revealed the role of dopaminergic transmission in hypofrontality.

Hypofrontality is a state of decreased cerebral blood flow (CBF) in the prefrontal cortex of the brain.

Hypofrontality is a symptom of numerous neurological diseases defined as reduced utilization of glucose and blood flow in the prefrontal cortex.

Like the other mental diseases that display hypofrontality, ADHD shows decreases in prefrontal cortex size and function.

With hypofrontality being linked to psychiatric diseases, depression, and drug addiction, there is a possibility that they all may have some common pathophysiological mechanism linking the diseases.

He concludes perceptively with a neurobiological discussion which equates the duality mentioned above with both hypofrontality and hyperactivity in posterior sites in schizophrenics.

Hypofrontality can be difficult to detect under resting conditions, but under cognitive challenges, it has been seen to correlate with memory deficits along with executive function deficits.

This 'hypofrontality' means low activity in the brain's frontal cortex, which is reportedly comparable to other altered states of consciousness, like drug-induced states, during meditation or when half-asleep.

Hypofrontality is also linked to an increase in norepinephrine transmission and decrease in dopaminergic transmission with reduced dopamine efflux in the frontal cortex.

Possible causes are hypothesized to be impaired synaptic connectivity and neurotransmission resulting from neurodevelopmental and/or genetic factors but there is not a complete understanding hypofrontality as a whole.

The working explanation of the neurophysiological mechanism behind hypofrontality is that hypofrontality is possibly caused by impaired synaptic connections, which results in diminished neurotransmission.

Hypofrontality is still not fully understood in its entirety, but there are a number of research projects that have been conducted, leading to progress in recognizing the signs of the symptom.

Hypofrontality is known to be a condition associated with the disorders listed below, though the exact role that hypofrontality plays in each of them has yet to be determined.