hypocapnia

The resulting low concentration of carbon dioxide in the blood is known as hypocapnia.

Even when marked, hypocapnia is normally well tolerated.

Shallow water blackout, for the mechanism behind self-induced hypocapnia.

She was hyperventilating, throwing herself into hypocapnia.

Hypercapnia is the opposite of hypocapnia.

Hyperventilation syndrome can cause respiratory alkalosis and hypocapnia.

Self-induced hypocapnia through hyperventilation is the basis for the deadly schoolyard fainting game.

There are two distinct methods used to achieve oxygen deprivation: strangulation and self-induced hypocapnia.

Paradoxically, hyperventilation, such as the fainting game, self-induced hypocapnia, or shallow water blackout.

The two mechanisms are strangulation and self-induced hypocapnia and work as follows:

The opposite condition is hyperventilation (too much ventilation), resulting in low carbon dioxide levels (hypocapnia), rather than hypercapnia.

Self-induced hypocapnia.

Hyperventilation artificially depletes this (CO) causing a low blood carbon dioxide condition called hypocapnia.

Excessive hypocapnia is readily identifiable as it causes dizziness and tingling of the fingers, refer to hyperventilation for details.

This condition is thought to result from severe hypocapnia that induces blood vessels in the brain to constrict, leading to brain ischemia.

Meanwhile, reduction of blood carbon dioxide levels (hypocapnia) can alter the blood pH and indirectly contribute to nervous system malfunctions.

This confusion is exacerbated by the fact that in the case of deep water blackout hypocapnia may be involved even if ascent is the actual precipitator.

One mechanism is that low carbon dioxide (hypocapnia) causes increased blood pH level (respiratory alkalosis), causing blood vessels to constrict.

The second mechanism requires hyperventilation (forced overbreathing) until symptoms of hypocapnia such as tingling, light-headedness or dizziness are felt, followed by a breath-hold.

In some versions the bear-hug is replaced by pressure on the neck in which case blackout is a hybrid of strangulation and self-induced hypocapnia.

Clinical fat embolism syndrome presents with tachycardia, tachypnea, elevated temperature, hypoxemia, hypocapnia, thrombocytopenia, and occasionally mild neurological symptoms.

However, hypocapnia causes cerebral vasoconstriction, leading to cerebral hypoxia and this can cause transient dizziness, visual disturbances, and anxiety.

Since carbon dioxide is carried as carbonic acid in the blood, hypocapnia results in the blood becoming alkaline, i.e. the blood pH value rises.

Because the brain stem regulates breathing by monitoring the level of blood CO, hypocapnia can suppress breathing to the point of blackout from cerebral hypoxia.

This leads to shifts in blood pH (respiratory alkalosis or hypocapnia), which in turn can lead to many other symptoms, such as paresthesia, dizziness, burning and lightheadedness.