hypersensitive response

Such compounds can be implied in the hypersensitive response of plants.

High levels of reactive oxygen species will cause the hypersensitive response.

Reactive oxygen species also play an important role in the hypersensitive response of plants against pathogen attack.

Hypersensitive response - a rapid host cell death response associated with defence mediated by "Resistance genes."

PVY induces hypersensitive responses in a wide range of potato cultivars.

Plant ETI often causes a hypersensitive response - a programmed cell death response.

The two proteins induced a hypersensitive response and systemic acquired resistance within the banana plant after being exposed to the bacterial pathogen.

A bean homolog of AtCP1 has been shown to be associated with the hypersensitive response [ 57].

A common example is hay fever, which is caused by a hypersensitive response by skin mast cells to allergens.

The hypersensitive response (HR) is a mechanism, used by plants, to prevent the spread of infection by microbial pathogens.

As the larvae migrate through the skin an inflammatory response, dermatitis, is often stimulated which can be exacerbated in hosts which give hypersensitive responses.

They are toxic proteins responsible for inducing a necrotic and systemic hypersensitive response in plants from the Solanaceae and Cruciferae families.

Food allergy and variable IgE response to food substances has been observed in some patients which implies role of hypersensitive response in pathogenesis.

It carries Mex-1 resistance gene, that causes Hypersensitive Response (HR) in the plant, also having low reproductive factor to M. exigua.

Some RXLR proteins are avirulence proteins, meaning that they can be detected by the plant and lead to a hypersensitive response which restricts the growth of the pathogen.

The first P. syringae type III effectors to be characterized were identified by their ability to cause a plant-type hypersensitive response upon recognition by plant resistance or R genes.

Recognition of a pathogen effector leads to a dramatic immune response known as the hypersensitive response, in which the infected plant cells undergo cell death to prevent the spread of the pathogen.

Analyzing one of Erwinia's hrp proteins, the harpin factor, Dr. Beer of Cornell has learned that it is a potent elicitor of the hypersensitive response, a plant's premier line of defense against a microbe.

When a part of a plant becomes infected, the plant produces a localized hypersensitive response, whereby cells at the site of infection undergo rapid apoptosis to prevent the spread of the disease to other parts of the plant.

These genes and their associated proteins are responsible for pathogen recognition and activation of defense signaling networks leading to the hypersensitive response, which is one of the mechanisms of the resistance of plants to pathogen infection.

Plants have two main immune responses-the hypersensitive response, in which infected cells seal themselves off and undergo programmed cell death, and systemic acquired resistance, where infected cells release signals warning the rest of the plant of a pathogen's presence.

Of keenest interest to them is the plant's earliest and most outstanding defense mechanism, the hypersensitive response, when the plant commands part of its own flesh to self-destruct and douse an invading organism with the toxic, acidic contents of the ruptured cells.

In pathogen interactions, the common short-term response is the hypersensitive response, in which cells surrounding the site of infection are signaled to undergo apoptosis, or programmed cell death, in order to prevent the spread of the pathogen to the rest of the plant.

They also respond by the localized activation of a cell-death program, designated the hypersensitive response (HR), and by the systemic activation of cellular and molecular defenses, termed systemic acquired resistance (SAR) [ 1 2 3 4 5 6 7 ] .

It is involved in such diverse functions as regulation of defense mechanisms in plant-pathogen interaction, promotion of the plant hypersensitive response, symbiosis (for example, with organisms in nitrogen-fixing root nodules), development of lateral and adventitious roots and root hairs, and control of stomatal opening.