hypercalciuria

Hypercalciuria or hypercalcinuria is the condition of elevated calcium in the urine.

High calcium in the urine (hypercalciuria).

Rice-bran treatment for calcium stone formers with idiopathic hypercalciuria.

Excess calcium in the urine (hypercalciuria) occurs due to increased bone demineralisation with acidosis.

It is also sometimes used for hypercalciuria, Dent's disease and Ménière's disease.

Physiological effects of slow release potassium phosphate for absorptive hypercalciuria: a randomized double-blind trial.

Renal colic is usually caused by pre-existing nephrolithiasis, as may occur in patients with chronic hypercalciuria.

Urinary stone formation (related to alkaline urine, hypercalciuria, and low urinary citrate).

Hyperoxaluria And other causes of hypercalcemia (and thus hypercalciuria)

Immobilization (leading to hypercalcemia and hypercalciuria)

Mutations in the ADCY10 gene are associated with an increased risk of adsorptive hypercalciuria.

Hypercalcaemia results in hypercalciuria, which can also be caused by Cushing's syndrome or hyperparathyroidism.

They are also commonly seen in people with underlying metabolic disorders, such as idiopathic hypercalciuria, hyperparathyroidism, and gout.

Treating co-existing hypercalciuria and hyperuricosuria will also be helpful in reducing hematuria.

Sustained reduction in urinary calcium during long-term treatment with slow release neutral potassium phosphate in absorptive hypercalciuria.

The mechanisms by which CLC-5 dysfunction results in hypercalciuria and the other features of Dent's disease remain to be elucidated.

Amiloride also increases distal tubular calcium reabsorption and has been used as a therapy for idiopathic hypercalciuria.

Because of its rather rare occurrence, Dent's disease is often diagnosed as idiopathic hypercalciuria, i.e., excess calcium in urine with undetermined causes.

Because of their promotion of calcium retention, thiazides are used in the treatment of Dent's Disease or idiopathic hypercalciuria.

Thiazides are useful for treating absorptive hypercalciuria, a condition in which high urinary calcium is a result of excess absorption from the gastrointestinal tract.

An abnormally high amount of urinary calcium is called hypercalciuria and an abnormally low amount is called hypocalcuria.

This condition may be aggravated by high levels of estradiol and prolactin, such as in pregnancy, leading to hypercalciuria and/or compensatory hypoparathyroidism.

Rabbits are prone to hypercalciuria due to intestinal absorption of calcium not being dependent on vitamin D and a high fractional urinary excretion of calcium.

Mutations in this gene have been associated with antenatal Bartter syndrome, which is characterized by salt wasting, hypokalemic alkalosis, hypercalciuria, and low blood pressure.

About 85% of infants dispose of excess amounts of calcium in the urine (hypercalciuria) and kidneys (nephrocalcinosis), which may lead to kidney stones.