human placental lactogen

For information on the human form, see Human placental lactogen.

This is likely due to pregnancy related factors such as the presence of human placental lactogen that interferes with susceptible insulin receptors.

Increased levels of certain hormones (including cortisol, estrogen, and human placental lactogen) interfere with the ability of insulin to manage blood sugar.

Additionally, human placental lactogen (hPL) is produced by the placenta, ensuring nutrient supply to the fetus.

It may secrete human placental lactogen (human chorionic somatomammotropin), and result in a false-positive pregnancy test.

Cortisol and progesterone are the main culprits, but human placental lactogen, prolactin and estradiol contribute, too.

An enhancer for the human placental lactogen gene is found 2 kb downstream of the gene and participates in the cell-specific control gene expression.

Human placental lactogen (hPL), also called human chorionic somatomammotropin (HCS), is a polypeptide placental hormone.

Human placental lactogen (HPL) - From the second month of pregnancy, the placenta releases large amounts of HPL.

Maturing into type 3 and then reaching full differentiation as type 4 lobules requires an increase of human placental lactogen (hPL) which occurs in the last few months of pregnancy.

Human placental lactogen (hPL) is produced by the placenta and stimulates lipolysis and fatty acid metabolism by the woman, conserving blood glucose for use by the fetus.

The syncytiotrophoblast secretes progesterone in addition to human chorionic gonadotropin (hCG) and human placental lactogen (HPL); hCG prevents degeneration of the corpus luteum.

Human placental lactogen is present in the tumor cells, while immunoperoxidase staining for human chorionic gonadotropin (hCG) is positive in only scattered cells, and serum hCG is relatively low.