heterozygote

It seems that some kind of heterozygote advantage is operating.

Thus you can tell that each allele is present in the heterozygote.

Cases of heterozygote advantage have been demonstrated in several organisms, including humans.

There, the heterozygote has the advantage in fitness.

Resistance to the following have all been proposed as possible sources of heterozygote advantage:

Merle is actually a heterozygote of an incompletely dominant gene.

The heterozygote is resistant to the malarial parasite which kills a large number of people each year.

The heterozygote has a permanent advantage (a higher fitness) wherever malaria exists.

When disease occurs because of two unrelated mutations, the patient is said to be a compound heterozygote.

The black form may be an example of heterozygote advantage; breeding in captivity is not yet conclusive on this.

If instead the two alleles are different, the individual is a heterozygote and is heterozygous.

In a heterozygote the effect of one allele may completely 'mask' the other.

The two major and most studied are heterozygote advantage and frequency-dependent selection.

A heterozygote may eventually show conspicuous leopard spots.

The specific case of heterozygote advantage due to a single locus is known as overdominance.

This effect is called heterozygote advantage.

Recent models suggest that a high number of alleles is implausible via heterozygote advantage alone.

The heterozygote frequency is kept high because of the fierce selection against both the homozygotes.

This is sufficient to maintain the system despite the fact that in this case the heterozygote has slightly lower viability.

When allele frequencies differ, the conditions for population heterozygote advantage may become broader or narrower.

These authors found the heterozygote state was indistinguishable from the noncarrier state.

In that case, heterosis is simply the result of the increased expression of heterozygote advantage.

As a consequence, the heterozygote genotype is selectively favored in areas with a high incidence of malaria.

The problem we have described with measuring population heterozygote advantage is not in principle limited to susceptibility/resistance studies of infectious diseases.

Also, there are some loci where the heterozygote is inherently fitter than either homozygote.