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There are no documented cases in which both types of gonadal tissue function.
Over a period of weeks, gonadal androgen production is greatly reduced.
During menopause, there are drastic changes in the production of gonadal hormones.
Growth and gonadal development may also be affected.
"But we're quite confident we have destroyed gonadal tissue."
The use of gonadal hormones pre-dates their identification and isolation.
Laboratory studies have found that long day illumination and high humidity trigger gonadal growth.
After gonadal maturation, females tend to grow faster than males.
Stage 5 typically represents maximal gonadal growth and adult hormone levels.
The germ cells split into two populations and move to the paired gonadal ridges.
The first type of gonadal dysgenesis discovered was Turner syndrome.
The cords were formed from the gonadal ridge.
The gonadal ridge, in turn, develops into a gonad.
Those with hematologic or gonadal tumors would be at greatest risk for this eventuality.
Male gonadal toxicity is a complex issue in Hodgkin lymphoma.
Gonadal germ cell tumors form in the testicles or ovaries.
This protein may be involved in both male gonadal differentiation and perineurial development.
In winter and spring consecutive spawns take place, interrupted by gonadal restoration periods.
They influence gonadal hormone secretion, which for example.
However, some cells of mesonephric origin join the gonadal ridge.
Gonadal origin should be excluded by careful testicular examination and ultrasound.
Increases in gonadal estrogen promote breast development, female fat distribution and skeletal growth.
Once they have reached the gonadal ridge they are called oogonia.
Then the production of gonadic cells begins again, progresses through the new moon, and reaches its maximum at the full moon when the ova and the spermatozoa are mature.'