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The test measures levels of a brain chemical called glutamate.
The mechanism is not well known, but may be related to effects on glutamate activity.
Just five minutes of excess glutamate is enough to kill neurons.
High glutamate levels have been tied to mood disorders in several studies.
The price fixing, he said, related to a food product known as sodium glutamate.
They found that the drug prevents glutamate from killing neurons.
Glutamate also plays a role in learning and memory.
Brain cells in people with Alzheimer's disease release too much glutamate.
But when the gene's activity is disrupted, levels of glutamate may build up.
Alcohol has a powerful effect on glutamate as well.
Under normal conditions, he said, glutamate is bound up tightly inside cells.
Nevertheless, many researchers believe that glutamate is the leading agent of cell death.
Drugs which interact with the other types of glutamate receptor are without effect.
It is used in scientific research into the function of the glutamate transporters.
It is also known to maintain high internal glutamate concentrations under stress conditions.
Calcium can also cause the release of more glutamate.
Glutamate is another neurotransmitter that tends to decrease with age.
Rather, they focus on the role glutamate naturally plays in the brain, and how it can be disrupted.
High pressure in the brain and low oxygen are thought to set off the overproduction of glutamate.
Glutamate also plays an important role in the body's disposal of excess or waste nitrogen.
"There was a clear difference in glutamate levels between ischemic stroke patients and all the other groups," she says.
Mice lacking functional glutamate receptor 1 were reported in 1994.
A conserved glutamate residue is thought to contribute to a metal center.
Excessive glutamate release is a known major cause of neuronal cell death.
Glutamate binds at the bottom of the active site.