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The fluid filtered in this way is called glomerular filtrate.
Thus, the glomerular filtrate becomes more concentrated, which is one of the steps in forming urine.
According to this theory, that charge plays a major role in the selective exclusion of albumin from the glomerular filtrate.
Glomerular capillaries have a reflection coefficient close to 1 as normally no protein crosses into the glomerular filtrate.
One unique feature of pronephroi is the arrangement by which the glomerular filtrate is generated and collected by the nephron.
The glomerular filtrate flows directly into the coelom, or a dorsal compartment of the coelom known as the nephrocoel.
Fluids from blood in the glomerulus are collected in the Bowman's capsule (i.e., glomerular filtrate) and further processed along the nephron to form urine.
A blood level of approximately 180 mg/dL is the renal glucose threshold below which all glucose is reabsorbed from glomerular filtrate.
These solutes are reabsorbed isotonically, in that the osmotic potential of the fluid leaving the proximal convoluted tubule is the same as that of the initial glomerular filtrate.
The kidneys may be irreversibly compromised if the concentration of calcium in the glomerular filtrate exceeds its solubility, resulting in calcium precipitation in the renal tubules (nephrocalcinosis).
If a substance has passed through the glomerular capillary endothelial cells, glomerular basement membrane, and podocytes, then it enters the lumen of the tubule and is known as glomerular filtrate.
In physiology, reabsorption or tubular reabsorption is the flow of glomerular filtrate from the proximal tubule of the nephron into the peritubular capillaries, or from the urine into the blood.
Renal tubular acidosis (proximal type) (Fanconi syndrome) occurs when the PTECs are unable to properly reabsorb glomerular filtrate so that there is increased loss of bicarbonate, glucose, amino acids, and phosphate.
Kidneys play a very large role in human osmoregulation by regulating the amount of water reabsorbed from glomerular filtrate in kidney tabules, which is controlled by hormones such as antidiuretic hormone (ADH), aldosterone, and angiotensin II.
Renal cell carcinoma (RCC, also known as hypernephroma) is a kidney cancer that originates in the lining of the proximal convoluted tubule, the very small tubes in the kidney that transport GF (glomerular filtrate) from the glomerulus to the descending limb of the nephron.