gastric epithelium

Unidentified curved bacillus on gastric epithelium in active chronic gastritis.

The bacterium, however, does not penetrate the gastric epithelium and the mechanism by which it induces inflammation of the underlying mucosa is unknown.

The cellular damage caused by thermal stress could lead the esophagus and gastric epithelium to be metaplasic, adapting to the chronic injury.

In the gastrointestinal tract, the gastric epithelium expresses very low levels of EpCAM.

We proposed previously that the three patterns of physical relation between H pylori and gastric epithelium could represent stages in evolution of bacterial infection.

The question has been raised whether these changes result in restoration of the duodenal mucosa and disappearance of gastric epithelium from the duodenal bulb.

The Table shows the relation of the number of micro-organisms with both bacterial morphology and their modes of contact with gastric epithelium.

Gastrin is transferred from a specific type of G cell in the gastric epithelium to the ECL cells by blood.

We now conclude that two ultrastructural bacterial patterns exist, and that these two patterns are linked to the modes of contact with gastric epithelium.

The O antigen of LPS may be fucosylated and mimic Lewis blood group antigens found on the gastric epithelium.

Duodenal biopsy specimens were stained with periodic acid Schiff (PAS) for the detection of metaplastic gastric epithelium.

As this virus can cause thrombocytopenia, injudicious use of these drugs can cause erosions in the gastric epithelium leading to exsanguinating upper GI bleed (due to thrombocytopenia).

The integrity of the gastric epithelium depends on a balance between aggressive and defensive factors include the mucus layer and the secretion of bicarbonate to neutralise gastric hydrochloric acid.

An increased proliferative capacity of the gastric epithelium next to the areas of MALT derived lymphomas has been postulated (P G Isaacson, personal communication) as a possible mechanism.

In the case of H. contortus the dauer nematode can remain in the gastric epithelium for months and until the right signals indicating conditions outside the host are favorable for egg survival, it will then trigger resumption of development.

Subsequently, it has been suggested that gastric metaplasia in the duodenum and H pylori associated gastritis might be synergistic in the pathogenesis of duodenitis, with the metaplastic gastric epithelium allowing H pylori to colonise the duodenal mucosa, where it produces an acute inflammatory response.