gastric acidity

Gastric acidity influences the blood response to a beta-carotene dose in humans.

Individuals who take medications for depression, gastric acidity or antihistamines should not undergo this test.

Zollinger-Ellison syndrome, which can be present with increased gastric acidity due to gastrin production.

Gastric acidity is responsible for the destruction of the majority of ingested bacteria.

Almagate, indicated for gastric acidity which has become the antacid of reference in the Spanish market.

It was trialled as a treatment for excessive gastric acidity, but withdrawn after human trials showed liver damage.

Drugs that decrease gastric acidity or sucralfate should be administered 2 hrs after ketoconazole.

Likewise, gastric acidity converts sucrose to glucose and fructose during digestion.

This dose, however, is less in those with lowered gastric acidity (for instance those using proton pump inhibitors).

The mechanisms whereby smoking influences ulcer healing are unknown; it probably does not affect gastric acidity, although it may alter duodenal motility and acidity.

H pylori may cause transient hypochlorrhydria in acutely infected subjects but normal levels of gastric acidity have been observed in asymptomatic, presumably chronically infected subjects.

This observation will be followed by further direct measures of gastric acidity, but should stimulate renewed interest in the potentially crucial relationship between the gastric acid barrier and intestinal flora.

In addition to the reduction of gastric acidity, antacids also alter the profile of prostaglandins produced by gastroduodenal mucosa and this may promote mucosal healing and be related to its therapeutic effect.

Gastric juice may be a significant source for the daily loss of iron since it is present in gastric juice at about 3.5 M, but correlation with gastric acidity or comparison between normal and ulcer patients could not be undertaken because of contamination.

In these patients gastric acidity does not affect faecal pancreatic isoamylase activity, but faecal salivary type isoamylase activity is raised in patients with gastric hypoacidity, indicating the important role of H 2 receptor antagonist administration with pancreatic supplements.