favism

This potentially fatal condition is called favism after the fava bean.

However, not all individuals with G6PD deficiency show favism.

Other than this, the specifics of the chemical relationship between favism and G6PD are not well understood.

People who do not have enough have a disease called G6PD deficiency (or favism).

The reason is unclear: perhaps the flatulence they cause, perhaps as protection from potential favism, but most likely for magico-religious reasons.

These results suggest that divicine is a direct-acting hemolytic agent and likely the direct cause of favism.

In Mediterranean countries such individuals can develop a hemolytic diathesis (favism) after consuming fava beans.

Vicine is toxic, causing the disease favism, in individuals who have a hereditary loss of the enzyme glucose-6-phosphate dehydrogenase.

Causes hemolytic anemia in carriers of the G6PD (favism) enzymatic deficiency.

Symptoms of favism include hemolysis, prolonged jaundice, kernicterus, and even acute renal failure in extreme cases.

The seeds of bitter melon contains vicine, so can trigger symptoms of favism in susceptible individuals.

Commonly known as "favism", as it can be triggered by chemicals existing naturally in broad (or fava) beans.

Glucose-6-phosphate dehydrogenase deficiency (sometimes also called G6PD deficiency, or favism) is a hereditary disease.

"Exactly, like favism among certain Mediterranean families or 'cold-fat disease' among the Maroon tribes of Venezuela."

One of the most common diseases among certain ethnic groups in Iran was favism, a metabolic disease caused by an enzyme deficiency in red blood cells.

Glucose-6-phosphate dehydrogenase deficiency (G6PD or favism)

Fava beans - rarely toxic to people, especially males, of Cretan origin, susceptible to favism, a rare genetic form of fatal hemolytic anemia.

Metabolic food reactions are due to an inborn or acquired errors of metabolism of nutrients such as in diabetes melitus, lactase deficiency, phenylketonuria and favism.

Glucose-6-phosphate dehydrogenase (G6PD) deficiency: People with G6PD deficiency might develop "favism" after eating bitter melon seeds.

One theory for the Pythagoreans' avoidance of beans is avoidance of favism, but more likely, this was a philosophical matter, such as the belief that beans and humans were created from the same material.

G6PD deficiency is closely linked to favism, a disorder characterized by a hemolytic reaction to consumption of broad beans, with a name derived from the Italian name of the broad bean (fava).

The name favism is sometimes used to refer to the enzyme deficiency as a whole, although this is misleading as not all people with G6PD deficiency will manifest a physically observable reaction to consumption of broad beans.

Moreover, common vetch as well as broad bean - and probably other species of Vicia too - contain oxidants like covicine, isouramil, divicine and vicine in quantities sufficient to lower glutathione levels in G6PD-deficient persons to cause favism disease.

Divicine has been deemed a hemotoxic component of fava beans and plays a role in the development of favism, a disorder that involves a hemolytic response to the consumption of broad beans due to glucose-6-phosphate dehydrogenase (G6PD or G6PDH) deficiency.

Some, for example Cicero, say perhaps the flatulence caused by beans is an emergency response system, as protection from potential favism, perhaps because they resemble the kidneys and genitalia, but most likely for magico-religious reasons, such as the belief that beans and human beings were created from the same material.