Familial adenomatous polyposis: There is some concern that fish oil might further increase the risk of getting cancer in people with this condition.

Patients with familial adenomatous polyposis have a uniformily excellent functional result.

Article: Psychological implications of living with familial adenomatous polyposis.

The familial adenomatous polyposis gene is a tumor suppressor, for example.

Some researchers believe that Turcot syndrome is a variant of familial adenomatous polyposis.

Familial adenomatous polyposis can have different inheritance patterns and different genetic causes.

They also raise into question the efficacy of aspirin as a chemotherapeutic for colon cancer, and perhaps more specifically, familial adenomatous polyposis.

Inherited conditions such as familial adenomatous polyposis and hereditary non-polyposis colon cancer Obesity.

Familial adenomatous polyposis.

Petersen GM: Genetic testing and counseling in familial adenomatous polyposis.

Then company scientists demonstrated that Celebrex could suppress polyp formation in patients with familial adenomatous polyposis.

Polyposis registries exist for the purpose of understanding the genetic disease familial adenomatous polyposis.

Paired sigmoidoscopic biopsy specimens were obtained from 17 control patients and five patients with familial adenomatous polyposis.

Jagelman DG: Clinical management of familial adenomatous polyposis.

Most cases are sporadic, but some are associated with familial adenomatous polyposis (FAP).

One specimen from each patient was examined histologically to exclude mucosal disease, notably microadenoma in familial adenomatous polyposis.

Having familial adenomatous polyposis (FAP).

In this disorder, familial adenomatous polyposis, the patient's large intestine, or colon, becomes virtually carpeted with polyps, some of which become malignant.

Upper gastrointestinal tract malignancy is a major cause of death in patients with familial adenomatous polyposis (FAP).

In colon cancer, familial adenomatous polyposis is very strongly associated with the FAP gene, but it is a rare defect.

Frequency of defaecation is usually less than three per 24 hours in patients with familial adenomatous polyposis but remains variable in those with ulcerative colitis.

Using this technique, Spigelman et al have examined DNA extracted from the foregut of patients with familial adenomatous polyposis.

Both copies of the MYH gene are mutated in individuals who have autosomal recessive familial adenomatous polyposis.

The most common mutation in familial adenomatous polyposis is a deletion of five bases in the APC gene.

Pouchitis has not occurred in patients with constipation or megacolon but has been found in three of 24 patients with familial adenomatous polyposis.