enterohepatic circulation

Due to the effect of enterohepatic circulation a second peak is seen several hours later.

This is interpreted by the authors as evidence that no enterohepatic circulation takes place.

Some of it is recycled through the enterohepatic circulation.

This replaced bile acid loss from the enterohepatic circulation.

This process is known as enterohepatic circulation.

Drugs may remain in the enterohepatic circulation for a prolonged period of time as a result of this recycling process.

By doing so, they bind bile acids and sequester them from enterohepatic circulation.

Also known as the enterohepatic circulation.

This enterohepatic circulation contributes to maintaining estradiol levels.

It is classified as a second generation sulfonylurea, which means that it undergoes enterohepatic circulation.

This mechanism might facilitate bile salt absorption into the enterohepatic circulation by slowing intestinal transit time.

By inhibiting the enterohepatic circulation, more L-thyroxine will be lost through defecation, thus lowering body thyroxine levels.

It undergoes enterohepatic circulation.

Enterohepatic circulation also means that some molecules which would not otherwise be very toxic can become extremely hepatotoxic as they reach unexpectedly high hepatic concentrations.

Repeated doses of activated carbon may be helpful by absorbing any toxins that are returned to the gastrointestinal tract following enterohepatic circulation.

This input mimics the case of an intestinally absorbed solute that is excreted by the liver and then reabsorbed (enterohepatic circulation).

Sodium/bile acid cotransporters are integral membrane glycoproteins that participate in the enterohepatic circulation of bile acids.

They disrupt the enterohepatic circulation of bile acids by combining with bile constituents and preventing their reabsorption from the gut.

Based on kinetic studies, this increased fecal loss appears to be secondary to an interrupted enterohepatic circulation of bile acids [ 20 21 ] .

Individually, wide variations exist in the overall absorption process, and can be further modified by drugs (i.e. antibiotics) that affect the enterohepatic circulation or liver enzymes.

That long-lasting action was most likely due to slow dissociation of the enzyme-inhibitor complex and an active oxide metabolite that undergoes enterohepatic circulation.

Instead the conjugated bilirubin is converted back into the unconjugated form by the enzyme glucuronidase and a large proportion is reabsorbed through the enterohepatic circulation.

Since bile acids are made from endogenous cholesterol, the enterohepatic circulation of bile acids may be disrupted to lower cholesterol.

All four of these bile acids can be taken back up into the blood stream, return to the liver, and be re-secreted in a process known as enterohepatic circulation.