duodenal ulcer disease

We conducted such a study in patients with active duodenal ulcer disease.

Therefore,H pylori treatment is recommended for patients with relapsing duodenal ulcer disease.

Duodenal ulcer disease has traditionally been associated with excess secretion of gastric acid.

All patients were volunteers and all had had previous vagotomy for benign duodenal ulcer disease.

Epigastric pain was the main clinical symptom of duodenal ulcer disease: this was experienced by all patients before entering the study.

This study suggests that both acid induced injury and H pylori infection may play a part in the pathogenesis of duodenal ulcer disease.

H pylori therapy is recommended in patients with relapsing duodenal ulcer disease, but a simple and safe treatment schedule is not yet available.

It is now widely accepted that H pylori is one of the most important factors in the pathogenesis of duodenal ulcer disease.

In 80%, the cause of choledochoduodenal fistula is usually penetrating duodenal ulcer disease in patients with a long ulcer history.

We therefore studied acid secretory responses to gastrin in a series of patients with duodenal ulcer disease before and after treatment to eradicate H pylori.

Thus H pylori infection combined with active ulceration may cause the increased basal acid secretion seen in duodenal ulcer disease.

These include differences in the prevalence of duodenal ulcer disease, consumption of alcohol, and the use of anti-secretory drugs in different populations.

Thus, amoxicillin plus omeprazole should be recommended as the treatment of choice in eradicating H pylori in patients with duodenal ulcer disease.

Thus, in addition to duodenal ulcer disease,H pylori eradication may also cure gastric ulcer disease.

The role of the increased serum gastrin concentration induced by H pylori in the pathogenesis of duodenal ulcer disease is also unknown.

H pylori was not examined because its importance in duodenal ulcer disease was not widely recognised when this study was being planned.

Indeed, there were fewer patients than expected taking NSAIDs, perhaps since intake had been reduced because of chronic duodenal ulcer disease.

The finding of enhanced fasting gastrin concentrations in H pylori positive subjects and in duodenal ulcer disease cannot easily be explained.

Risk factors for delayed duodenal ulcer healing are mentioned in all textbooks as important considerations in the management of duodenal ulcer disease.

It is widely held that the increased basal acid secretion in patients with duodenal ulcer disease is determined by vagal drive rather than circulating gastrin.

Eight healthy volunteers considered free from peptic ulcer disease (controls) and eight patients with endoscopically proved duodenal ulcer disease were studied.

In 1992, a first controlled study showed that H pylori positive gastric ulcer disease will respond in exactly the same way as duodenal ulcer disease does.

Hyperpepsinogenaemia A has been shown in patients with duodenal ulcer disease, Zollinger-Ellison syndrome, hypertrophic gastropathy, chronic renal failure, and during omeprazole treatment.

Helicobacter pylori (H pylori ) eradication heals chronic active type B gastritis and dramatically changes the natural history of duodenal ulcer disease.