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Granule cells in the dorsal cochlear nucleus play a role in the perception and response to sounds in our environment.
Fusiform cells (also known as pyramidal cells) are found in the dorsal cochlear nucleus (DCN).
Its axon projects to the molecular layer of the dorsal cochlear nucleus where it forms parallel fibers, also similar to cerebellar granule cells.
The dorsal cochlear nucleus (DCN, also known as the "tuberculum acousticum"), is a cortex-like structure on the dorso-lateral surface of the brainstem.
The only one found in mammals is the dorsal cochlear nucleus (DCN), one of the two primary sensory nuclei that receive input directly from the auditory nerve.
The granule cells in the dorsal cochlear nucleus are small neurons with two or three short dendrites that give rise to a few branches with expansions at the terminals.
They have also been described and studied in layers V of the entorhinal cortex, the inferior olive in vivo, the olfactory bulb and the dorsal cochlear nucleus.
The fusiform cells of the Dorsal cochlear nucleus, which are thought to contribute to localization in elevation, bypass the SOC and project directly to the inferior colliculus.
The inferior colliculus is the first place where vertically orienting data from the fusiform cells in the dorsal cochlear nucleus can finally synapse with horizontally orienting data.
The dorsal cochlear nucleus (DCN) contains type IV cells which are particularly excited by noises containing spectral notches, implicating them in the processing of vertical sound localization.
In mammals, this region is anatomically and physiologically split into two regions, the dorsal cochlear nucleus (DCN), and ventral cochlear nucleus (VCN).
Axons from the spiral ganglion cells of the lower frequencies innervate the lateral-ventral portions of the dorsal cochlear nucleus and the ventrolateral portions of the anteroventral cochlear nucleus.
This has led to the suggestion that one cause of tinnitus might be a homeostatic response of central dorsal cochlear nucleus auditory neurons that makes them hyperactive in compensation to auditory input loss.
Models of hearing loss and the brain support the idea a homeostatic response of central dorsal cochlear nucleus neurons could result in them being hyperactive in a compensation process to the loss of hearing input.
In contrast, the axons from the higher frequency organ of corti hair cells project to the dorsal portion of the anteroventral cochlear nucleus and the dorsal-medial portions of the dorsal cochlear nucleus.
The superior olivary complex is located in the pons, and receives projections predominantly from the ventral cochlear nucleus, although the dorsal cochlear nucleus projects there as well, via the ventral acoustic stria.
The dorsal cochlear nucleus (DCN) has input from the LL and output to the contralateral LL via the ipsilateral and contralateral Dorsal Acoustic Stria.
Axons leaving the dorsal cochlear nucleus (DCN) form the dorsal acoustic stria, which reaches primarily the contralateral dorsal nucleus of the lateral lemniscus and the central nucleus of the inferior colliculus.
The central nucleus of the IC is a nearly obligatory relay in the ascending auditory system, and most likely acts to integrate information (specifically regarding sound source localization from the superior olivary complex and dorsal cochlear nucleus) before sending it to the thalamus and cortex.
The fibers of the auditory nerve innervate both cochlear nuclei in the brainstem, the cochlear nucleus magnocellularis (mammalian anteroventral cochlear nucleus) and the cochlear nucleus angularis (see figure; mammalian posteroventral and dorsal cochlear nuclei).