It has been shown that diploidy is restored by terminal fusion.

Due to the diploidy of humans (and most animals), there are two alleles for any given gene.

Cdk-1 is believed to regulate the G2 checkpoint of the cell cycle and is required to maintain diploidy.

In other cases, collectively called automictic parthenogenesis, the ploidy is restored to diploidy by various means.

By preventing segregation of chromosomes in the first mitotic division, diploidy is restored, which leads to the development of a parthenogenetic female.

All chromosomes (uniparental diploidy)

We therefore determined the frequencies of spore viability and meiotic diploidy as genetic measures of chromosome segregation errors.

The rice and the wheat genomes, for example, can have 4-6 copies of whole genomes (wheat) whereas animals typically only have 2 (diploidy).

Because diploid spores could contain complementing markers and be mistaken for recombinants, we tested all spore colonies for diploidy by replica plating on complete media containing Phloxin-B (YEA-B).

The Law of Segregation states that every individual possesses a pair of alleles (assuming diploidy) for any particular trait and that each parent passes a randomly selected copy (allele) of only one of these to its offspring.

The mathematical biologist Ronald Fisher analysed the contribution that having two copies of each gene (diploidy) would make to honest signalling, demonstrating that a runaway effect could occur in sexual selection, depending sensitively on the balance of costs and benefits.

Children younger than 1 year at diagnosis have a good chance of long-term survival (i.e., a 5-year disease-free survival rate of 50%-80%),[11,12] with outcome particularly dependent on MYCN amplification and tumor cell ploidy (e.g., hyperdiploidy confers a favorable prognosis while diploidy predicts early treatment failure).