The entire process of blood vessel escape is known as diapedesis.

Infection seems to be correlated to haemocyte destruction and diapedesis.

This impairs the ability of the leukocytes to stop and undergo diapedesis.

IL-8 presumably plays an important part in neutrophil diapedesis through vascular endothelium and focal recruitment at inflamed sites.

In the case of inflammation, it refers to the movement of white blood cells from the capillaries to the tissues surrounding them (diapedesis).

Proestrus bleeding in dogs is common and is believed to be caused by diapedesis of RBCs from the blood vessels due to the increase of the estradiol-17β hormone.

Migration across the endothelium, known as transmigration, via the process of diapedesis: Chemokine gradients stimulate the adhered leukocytes to move between endothelial cells and pass the basement membrane into the tissues.

These cytokines increase the expression of adhesion factors on endothelial cells to enable transmigration (also called diapedesis) of immunocompetent cells, such as phagocytes, lymphocytes and others, to sites of infection.

Stricker is remembered for his extensive studies in the fields of histology and experimental pathology, and is credited with making discoveries involving the diapedesis of erythrocytes and the contractility of vascular walls.