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If large enough, this depolarization results in an action potential.
There are several ways in which this depolarization can occur.
Only when there is a depolarization do these Ca channels open.
The point at which depolarization stops is called the peak phase.
Although the channel can still be activated, a much larger depolarization is needed.
This squeezing produced no current until five minutes in when a large depolarization was observed.
This depolarization, if it reaches a certain threshold, will cause an action potential.
This is also known as a wave of depolarization.
An important step in childhood development is the gradual depolarization of these two drives.
This spontaneous depolarization is due to the special phase 4 as described above.
This relatively slow depolarization continues until the threshold potential is reached.
In this way a wave of depolarization travels along the entire membrane.
This allows it to produce sustained Ca entry upon depolarization.
The larger the stimulus, the greater the depolarization, or attempt to reach threshold.
The depolarization of the membrane allows calcium channels to open as well.
It represents the smallest possible depolarization which can be induced in a muscle.
In neurons and some other cells, a large enough depolarization may result in an action potential.
This is because the current changes direction too quickly to trigger depolarization of nerve membranes.
This change makes the activation gate open more easily by low voltage depolarization.
This, in turn, leads to the neuron's depolarization and the influx of more Ca.
Depolarization of the membrane may also trigger these bursts.
These are the results of both slowed depolarization and repolarization.
For example, depolarization of the plasma membrane appears to be an important step in programmed cell death.
The signal strength was actually fading compared to the disruptor depolarization toward the end.
The membrane depolarization, in turn, leads to a sodium-dependent action potential at that location.