dentate nucleus

The dentate nucleus is not affected until late in disease progression.

The dentate nucleus is mostly responsible for planning and execution of fine movement.

The white matter of the cerebellum and the dentate nuclei are less often involved.

The role of the dentate nucleus can be described in two basic tenets:

Abnormalities are seen in the basal ganglia and dentate nucleus.

Cerebellar white matter and dentate nucleus lesions usually occur in children less than ten years of age.

Thus, there is a part of cerebellum that communicates exclusively with the dentate nucleus.

Similar abnormalities have been identified in the brainstem and cerebellar dentate nucleus.

During this time, the neurons of the dentate nucleus are similar in shape and form, being mainly bipolar cells.

The Purkinje cells send their afferents to the ipsilateral dentate nucleus.

These neurons are responsible for communication between the dentate nucleus and the cerebellar cortex.

The dentate nucleus is involved in basic circuitry work, including input to the cerebellum from everywhere else.

It also contains the emboliform-globose and dentate nuclei.

Calcifications in the caudate, dentate nuclei, putamen and thalami are also common.

Dentate nucleus axons can be divergent or convergent.

While the primary role of the dentate nucleus is control of the movement, it does play a role in sensory processing as well.

There is a near total loss of Purkinje fibers in the cerebellar hemisphere and an undetectable dentate nucleus.

The dentate nucleus sends commands and information to the motor and premotor areas in the forebrain.

In patients with degenerative diseases of dentate nucleus there is virtually a complete lack of BP.

Myelin edema is seen in the cerebellum, including the dentate nucleus, brain stem, and corticospinal tracts.

As a chunk of tissue, the dentate nucleus with overlying cerebellar cortex makes up a functional unit called the cerebrocerebellum.

There are three distinct routes from the dentate nucleus to the ventrolateral thalamus, including a direct, posterior, and anterior route.

During 22-28 weeks of gestation, which is the critical period in fetal development of the dentate nucleus, gyral formation occurs extensively over the entire surface.

Several pathological processes involve the dentate nucleus including metabolic, genetic, and neurodegenerative disorders, as well as some infections.

Toxic levels of metronidazole can cause symmetrical lesions in the brain in the corpus callosum and dentate nuclei.