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Most patients remain asymptomatic and corneal edema is usually absent.
Fluid buildup in the clear covering of the eye (corneal edema).
This prevents corneal edema, which gives it a cloudy appearance.
Symptoms mainly consist of decreased vision due to corneal edema.
The combination of high pressure within the eye (glaucoma) and corneal edema can result in reduced vision with pain.
Stromal keratitis manifests as a disc-shaped area of corneal edema.
Have a disease or abnormality of the cornea, such as keratoconus or corneal edema.
When splashed onto the eye chloropicrin has caused corneal edema and liquification of the cornea.
However, most dogs recover after a brief illness, although chronic corneal edema and kidney lesions may persist.
CAdV-1 was initially used in a vaccine for dogs, but corneal edema was a common complication.
Corneal edema can also occur as the result of compromised endothelial function due to intraocular inflammation or other causes.
Coloured halos are an indication of corneal edema, and are a warning that acute glaucoma may be present.
The typical infant who has congenital glaucoma usually is initially referred to an ophthalmologist because of apparent corneal edema.
Causes of media opacity include corneal edema, hyphema, cataract and vitreous hemorrhage.
In the late 1970s, intraocular lens implantation was often followed by severe corneal edema, due to endothelial cell damage during the surgery.
You have certain problems in the cornea, such as keratoconus, keratitis, corneal edema, or thinning of the cornea.
Corneal edema and signs of liver disease, such as jaundice, vomiting, and hepatic encephalopathy, may also occur.
Longstanding corneal edema leads to permanent scarring and is the major cause of decreased vision associated with HSV.
On ophthalmologic exam, one can detect increased intraocular pressure, distortion of the optic disc, and corneal edema, which manifests as haziness.
The commonly described triad of epiphora (excessive tearing), blepharospasm and photophobia may be missed until the corneal edema becomes apparent.
The mechanism behind contact lens-induced polymegathism is unknown, though it is also thought to be a byproduct of corneal edema and epithelial hypoxia.
Initial treatment may include hypertonic eyedrops and ointment to reduce the corneal edema and may offer symptomatic improvement prior to surgical intervention.
When used in ophthalmological procedures, sodium hyaluronate may cause postoperative inflammation, corneal edema or decompensation, and short-term increases in intraocular pressure.
The characteristic clinical findings are excrescences on a thickened Descemet membrane (cornea guttae), generalized corneal edema and decreased visual acuity.
Hence, patients with a history of Fuchs' dystrophy may be at a greater risk of corneal edema after ocular surgery as they have fewer functioning endothelial cells.